A cellular protein that competes with SV40 T antigen for binding to the retinoblastoma gene product.

E1A-binding region of the retinoblastoma gene product.

The retinoblastoma susceptibility gene product undergoes cell cycle-dependent dephosphorylation and binding to and release from SV40 large T.

Two distinct and frequently mutated regions of retinoblastoma protein are required for binding to SV40 T antigen.

Cloning of cDNAs for cellular proteins that bind to the retinoblastoma gene product.

Retinoblastoma gene product Rb accumulates during myogenic differentiation and is deinduced by the expression of SV40 large T antigen.

Interaction of DNA polymerase alpha-primase with cellular replication protein A and SV40 T antigen.

The binding site on SV40 DNA for a T antigen-related protein.

The regions of the retinoblastoma protein needed for binding to adenovirus E1A or SV40 large T antigen are common sites for mutations.

Definition of the minimal simian virus 40 large T antigen- and adenovirus E1A-binding domain in the retinoblastoma gene product.

Hyperphosphorylation of the retinoblastoma gene product is determined by domains outside the simian virus 40 large-T-antigen-binding regions.

E1A-binding domain of the retinoblastoma product can interact selectively with a sequence-specific DNA-binding protein.

Adenovirus E1a prevents the retinoblastoma gene product from complexing with a cellular transcription factor.

Three distinct effects of SV40 T-antigen gene transfection on cellular differentiation.

Identification of regions of the SV40 genome which contain preferred SV40 T antigen-binding sites.

Human papillomavirus type 16 E7 protein inhibits DNA binding by the retinoblastoma gene product.

Site-specific binding of wild-type p53 to cellular DNA is inhibited by SV40 T antigen and mutant p53.

Adenovirus E1a prevents the retinoblastoma gene product from repressing the activity of a cellular transcription factor.

Fluoxetine competes with cortisol for binding to human serum albumin.

Screening of human brain tumors for SV40-related T antigen.

Evidence for simian virus 40 (SV40) coding of SV40 T-antigen and the SV40-specific proteins in HeLa cells infected with nondefective adenovirus type 2-SV40 hybrid viruses.

Transformation of Bowes melanoma cells with SV40 T antigen.

Binding and unwinding--how T antigen engages the SV40 origin of DNA replication.

Interleukin-2-dependent phosphorylation of the retinoblastoma-susceptibility-gene product p110-115RB in human T-cells.

SV40 T-antigen-binding sites within the 5'-flanking regions of human U1 and U2 genes.

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E1A-binding region of the retinoblastoma gene product.

The retinoblastoma susceptibility gene product undergoes cell cycle-dependent dephosphorylation and binding to and release from SV40 large T.

Two distinct and frequently mutated regions of retinoblastoma protein are required for binding to SV40 T antigen.

Cloning of cDNAs for cellular proteins that bind to the retinoblastoma gene product.

Retinoblastoma gene product Rb accumulates during myogenic differentiation and is deinduced by the expression of SV40 large T antigen.

Interaction of DNA polymerase alpha-primase with cellular replication protein A and SV40 T antigen.

The binding site on SV40 DNA for a T antigen-related protein.

The regions of the retinoblastoma protein needed for binding to adenovirus E1A or SV40 large T antigen are common sites for mutations.

Definition of the minimal simian virus 40 large T antigen- and adenovirus E1A-binding domain in the retinoblastoma gene product.

Hyperphosphorylation of the retinoblastoma gene product is determined by domains outside the simian virus 40 large-T-antigen-binding regions.

E1A-binding domain of the retinoblastoma product can interact selectively with a sequence-specific DNA-binding protein.

Adenovirus E1a prevents the retinoblastoma gene product from complexing with a cellular transcription factor.

Three distinct effects of SV40 T-antigen gene transfection on cellular differentiation.

Identification of regions of the SV40 genome which contain preferred SV40 T antigen-binding sites.

Human papillomavirus type 16 E7 protein inhibits DNA binding by the retinoblastoma gene product.

Site-specific binding of wild-type p53 to cellular DNA is inhibited by SV40 T antigen and mutant p53.

Adenovirus E1a prevents the retinoblastoma gene product from repressing the activity of a cellular transcription factor.

Fluoxetine competes with cortisol for binding to human serum albumin.

Screening of human brain tumors for SV40-related T antigen.

Evidence for simian virus 40 (SV40) coding of SV40 T-antigen and the SV40-specific proteins in HeLa cells infected with nondefective adenovirus type 2-SV40 hybrid viruses.

Transformation of Bowes melanoma cells with SV40 T antigen.

Binding and unwinding--how T antigen engages the SV40 origin of DNA replication.

Interleukin-2-dependent phosphorylation of the retinoblastoma-susceptibility-gene product p110-115RB in human T-cells.

SV40 T-antigen-binding sites within the 5'-flanking regions of human U1 and U2 genes.

A cellular protein that competes with SV40 T antigen for binding to the retinoblastoma gene product.

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