A novel tetracyclic peptide, trapoxin, induces phenotypic change from transformed to normal in sis-oncogene-transformed NIH3T3 cells.

A novel compound, depudecin, induces production of transformation to the flat phenotype of NIH3T3 cells transformed by ras-oncogene.

Calcium oscillation associated with reduced protein kinase C activities in ras-transformed NIH3T3 cells.

Proteolytic enzymes in normal and transformed cells.

Single Synonymous Mutations in KRAS Cause Transformed Phenotypes in NIH3T3 Cells.

MyoD induces growth arrest independent of differentiation in normal and transformed cells.

Phenotypic modification of SV40-transformed hamster lymphoid cells in vivo.

OVA66, a tumor associated protein, induces oncogenic transformation of NIH3T3 cells.

CK2.1, a novel peptide, induces articular cartilage formation in vivo.

Potassium transport in normal and transformed mouse 3T3 cells.

Mobility of normal and virus-transformed cells in cellular aggregates.

Conditioned medium from ras oncogene-transformed NIH 3T3 cells induces bone resorption in vitro.

Depudecin: a novel compound inducing the flat phenotype of NIH3T3 cells doubly transformed by ras- and src-oncogene, produced by Alternaria brassicicola.

Organization of tubulin in normal and transformed rat kidney cells.

Regulation of differentiation in normal and transformed erythroid cells.

Mitogenic signal transduction in normal and transformed 32D hematopoietic cells.

The half-life of polyadenylated polysomal RNA from normal and transformed cells in monolayer culture.

Sulphated acid mucopolysaccharides in SV40-transformed human cells from normal and mucopolysaccharidosis patients.

SF2 activity in extracts from normal WI38 and transformed WI38VA13 cells.

s cyclin d2 induces cell death.

Injury induces a change in the functional characteristics of cells recovered from equine tendon.

Transformed BHK cells exhibiting normal or subnormal sugar uptake.

Phenotypic plasticity in normal breast derived epithelial cells.

A novel telomerase substrate precursor rapidly induces telomere dysfunction in telomerase positive cancer cells but not telomerase silent normal cells.

Peptide analysis of the transformation-specific antigen from avian sarcoma virus-transformed cells.

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A novel compound, depudecin, induces production of transformation to the flat phenotype of NIH3T3 cells transformed by ras-oncogene.

Calcium oscillation associated with reduced protein kinase C activities in ras-transformed NIH3T3 cells.

Proteolytic enzymes in normal and transformed cells.

Single Synonymous Mutations in KRAS Cause Transformed Phenotypes in NIH3T3 Cells.

MyoD induces growth arrest independent of differentiation in normal and transformed cells.

Phenotypic modification of SV40-transformed hamster lymphoid cells in vivo.

OVA66, a tumor associated protein, induces oncogenic transformation of NIH3T3 cells.

CK2.1, a novel peptide, induces articular cartilage formation in vivo.

Potassium transport in normal and transformed mouse 3T3 cells.

Mobility of normal and virus-transformed cells in cellular aggregates.

Conditioned medium from ras oncogene-transformed NIH 3T3 cells induces bone resorption in vitro.

Depudecin: a novel compound inducing the flat phenotype of NIH3T3 cells doubly transformed by ras- and src-oncogene, produced by Alternaria brassicicola.

Organization of tubulin in normal and transformed rat kidney cells.

Regulation of differentiation in normal and transformed erythroid cells.

Mitogenic signal transduction in normal and transformed 32D hematopoietic cells.

The half-life of polyadenylated polysomal RNA from normal and transformed cells in monolayer culture.

Sulphated acid mucopolysaccharides in SV40-transformed human cells from normal and mucopolysaccharidosis patients.

SF2 activity in extracts from normal WI38 and transformed WI38VA13 cells.

s cyclin d2 induces cell death.

Injury induces a change in the functional characteristics of cells recovered from equine tendon.

Transformed BHK cells exhibiting normal or subnormal sugar uptake.

Phenotypic plasticity in normal breast derived epithelial cells.

A novel telomerase substrate precursor rapidly induces telomere dysfunction in telomerase positive cancer cells but not telomerase silent normal cells.

Peptide analysis of the transformation-specific antigen from avian sarcoma virus-transformed cells.

A novel tetracyclic peptide, trapoxin, induces phenotypic change from transformed to normal in sis-oncogene-transformed NIH3T3 cells.

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