Alteration of voltage-dependence of Shaker potassium channel by mutations in the S4 sequence.

Hydrophobic substitution mutations in the S4 sequence alter voltage-dependent gating in Shaker K+ channels.

Biophysical and molecular mechanisms of Shaker potassium channel inactivation.

The isolated voltage sensing domain of the Shaker potassium channel forms a voltage-gated cation channel.

Specification of subunit assembly by the hydrophilic amino-terminal domain of the Shaker potassium channel.

Modulation of a Shaker potassium A-channel by protein kinase C activation.

Molecular basis for different rates of recovery from inactivation in the Shaker potassium channel family.

The Molecular Basis of Polyunsaturated Fatty Acid Interactions with the Shaker Voltage-Gated Potassium Channel.

Structural analysis of the S4-S5 linker of the human KCNQ1 potassium channel.

S3-S4 linker length modulates the relaxed state of a voltage-gated potassium channel.

Ligand-activated ion channels may share common gating mechanisms with the Shaker potassium channel.

Mutations in an S4 segment of the adult skeletal muscle sodium channel cause paramyotonia congenita.

On the activation-inactivation coupling in Shaker potassium channels.

Alteration of potassium channel gating: molecular analysis of the Drosophila Sh5 mutation.

Alterations in activation gating of single Shaker A-type potassium channels by the Sh5 mutation.

The inactivation gate of the Shaker K+ channel behaves like an open-channel blocker.

Cell-free expression of functional Shaker potassium channels.

Novel potassium channels encoded by the Shaker locus in Drosophila photoreceptors.

Suppression of the hERG potassium channel response to premature stimulation by reduction in extracellular potassium concentration.

Internal blockade of a Ca(2+)-activated K+ channel by Shaker B inactivating "ball" peptide.

Corrigendum: Mutations in the voltage-gated potassium channel gene KCNH1 cause Temple-Baraitser syndrome and epilepsy.

Mutations in the voltage-gated potassium channel gene KCNH1 cause Temple-Baraitser syndrome and epilepsy.

Gain-of-function mutations in potassium channel subunit KCNE2 associated with early-onset lone atrial fibrillation.

Two different types of potassium channels in human skeletal muscle activated by potassium channel openers.

A peptide from the Drosophila Shaker K+ channel inhibits a voltage-gated K+ channel in basolateral membranes of Necturus enterocytes.

Suggest Documents

Hydrophobic substitution mutations in the S4 sequence alter voltage-dependent gating in Shaker K+ channels.

Biophysical and molecular mechanisms of Shaker potassium channel inactivation.

The isolated voltage sensing domain of the Shaker potassium channel forms a voltage-gated cation channel.

Specification of subunit assembly by the hydrophilic amino-terminal domain of the Shaker potassium channel.

Modulation of a Shaker potassium A-channel by protein kinase C activation.

Molecular basis for different rates of recovery from inactivation in the Shaker potassium channel family.

The Molecular Basis of Polyunsaturated Fatty Acid Interactions with the Shaker Voltage-Gated Potassium Channel.

Structural analysis of the S4-S5 linker of the human KCNQ1 potassium channel.

S3-S4 linker length modulates the relaxed state of a voltage-gated potassium channel.

Ligand-activated ion channels may share common gating mechanisms with the Shaker potassium channel.

Mutations in an S4 segment of the adult skeletal muscle sodium channel cause paramyotonia congenita.

On the activation-inactivation coupling in Shaker potassium channels.

Alteration of potassium channel gating: molecular analysis of the Drosophila Sh5 mutation.

Alterations in activation gating of single Shaker A-type potassium channels by the Sh5 mutation.

The inactivation gate of the Shaker K+ channel behaves like an open-channel blocker.

Cell-free expression of functional Shaker potassium channels.

Novel potassium channels encoded by the Shaker locus in Drosophila photoreceptors.

Suppression of the hERG potassium channel response to premature stimulation by reduction in extracellular potassium concentration.

Internal blockade of a Ca(2+)-activated K+ channel by Shaker B inactivating "ball" peptide.

Corrigendum: Mutations in the voltage-gated potassium channel gene KCNH1 cause Temple-Baraitser syndrome and epilepsy.

Mutations in the voltage-gated potassium channel gene KCNH1 cause Temple-Baraitser syndrome and epilepsy.

Gain-of-function mutations in potassium channel subunit KCNE2 associated with early-onset lone atrial fibrillation.

Two different types of potassium channels in human skeletal muscle activated by potassium channel openers.

A peptide from the Drosophila Shaker K+ channel inhibits a voltage-gated K+ channel in basolateral membranes of Necturus enterocytes.

Alteration of voltage-dependence of Shaker potassium channel by mutations in the S4 sequence.

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