CORRECTION
Correction: The Extracellular Domain of Myelin Oligodendrocyte Glycoprotein Elicits Atypical Experimental Autoimmune Encephalomyelitis in Rat and Macaque Species The PLOS ONE Staff
Fig. 1 is an incorrect duplicate of Fig. 10. The publisher apologizes for the error. Please see the correct Fig. 1 here.
OPEN ACCESS Citation: The PLOS ONE Staff (2015) Correction: The Extracellular Domain of Myelin Oligodendrocyte Glycoprotein Elicits Atypical Experimental Autoimmune Encephalomyelitis in Rat and Macaque Species. PLoS ONE 10(2): e0117878. doi:10.1371/ journal.pone.0117878 Published: February 6, 2015 Copyright: © 2015 The PLOS ONE Staff. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
PLOS ONE | DOI:10.1371/journal.pone.0117878 February 6, 2015
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Figure 1. Booster immunizations with IgV-MOG in IFA caused a chronic course of atypical EAE. Seven Lewis rats were immunized with a mixture of 25 μg of GP69-88 and 50 μg of rat IgV-MOG in CFA on day 0. After resolution of a classical monophasic bout of EAE, the same rats were boosted with 200 μg of rat IgV-MOG in IFA on days 45 and 85. Rats were scored daily for clinical signs of EAE. After the IgV-MOG boosts in IFA, the incidence of chronic atypical EAE was 100% (7 of 7 rats). The red arrows (bottom panel) mark the dates of the boosts. Shown are the individual disease courses for the 7 rats. The blue line divides the clinical data into classical (left) and atypical (right) EAE courses. doi:10.1371/journal.pone.0117878.g001
PLOS ONE | DOI:10.1371/journal.pone.0117878 February 6, 2015
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Reference 1.
Curtis AD II, Taslim N, Reece SP, Grebenciucova E, Ray RH, et al. (2014) The Extracellular Domain of Myelin Oligodendrocyte Glycoprotein Elicits Atypical Experimental Autoimmune Encephalomyelitis in Rat and Macaque Species. PLoS ONE 9(10): e110048. doi: 10.1371/journal.pone.0110048 PMID: 25303101
PLOS ONE | DOI:10.1371/journal.pone.0117878 February 6, 2015
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Correction: The Extracellular Domain of Myelin Oligodendrocyte Glycoprotein Elicits Atypical Experimental Autoimmune Encephalomyelitis in Rat and Macaque Species The PLOS ONE Staff
Fig. 1 is an incorrect duplicate of Fig. 10. The publisher apologizes for the error. Please see the correct Fig. 1 here.
OPEN ACCESS Citation: The PLOS ONE Staff (2015) Correction: The Extracellular Domain of Myelin Oligodendrocyte Glycoprotein Elicits Atypical Experimental Autoimmune Encephalomyelitis in Rat and Macaque Species. PLoS ONE 10(2): e0117878. doi:10.1371/ journal.pone.0117878 Published: February 6, 2015 Copyright: © 2015 The PLOS ONE Staff. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
PLOS ONE | DOI:10.1371/journal.pone.0117878 February 6, 2015
1/3
Figure 1. Booster immunizations with IgV-MOG in IFA caused a chronic course of atypical EAE. Seven Lewis rats were immunized with a mixture of 25 μg of GP69-88 and 50 μg of rat IgV-MOG in CFA on day 0. After resolution of a classical monophasic bout of EAE, the same rats were boosted with 200 μg of rat IgV-MOG in IFA on days 45 and 85. Rats were scored daily for clinical signs of EAE. After the IgV-MOG boosts in IFA, the incidence of chronic atypical EAE was 100% (7 of 7 rats). The red arrows (bottom panel) mark the dates of the boosts. Shown are the individual disease courses for the 7 rats. The blue line divides the clinical data into classical (left) and atypical (right) EAE courses. doi:10.1371/journal.pone.0117878.g001
PLOS ONE | DOI:10.1371/journal.pone.0117878 February 6, 2015
2/3
Reference 1.
Curtis AD II, Taslim N, Reece SP, Grebenciucova E, Ray RH, et al. (2014) The Extracellular Domain of Myelin Oligodendrocyte Glycoprotein Elicits Atypical Experimental Autoimmune Encephalomyelitis in Rat and Macaque Species. PLoS ONE 9(10): e110048. doi: 10.1371/journal.pone.0110048 PMID: 25303101
PLOS ONE | DOI:10.1371/journal.pone.0117878 February 6, 2015
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