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© 1975 Nature Publishing Group
© 1975 Nature Publishing Group
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Phenobarbital increases spontaneous transmitter release at the frog neuromuscular junction.
Temperature-sensitive aspects of evoked and spontaneous transmitter release at the frog neuromuscular junction.
Clumping and oscillations in evoked transmitter release at the frog neuromuscular junction.
Transmitter release is evoked with low probability predominately by calcium flux through single channel openings at the frog neuromuscular junction.
Changes of quantal transmitter release caused by gadolinium ions at the frog neuromuscular junction.
Inhibitory effects of cholinergic agents on the release of transmitter at the frog neuromuscular junction.
The effects of tetraphenylboron on spontaneous transmitter release at the frog neuromuscular junction.
Effect of the "calcium ionophore" A-23187 on transmitter release at the frog neuromuscular junction.
The action of lindane in accelerating the spontaneous release of transmitter at the frog neuromuscular junction.
Augmentation: A process that acts to increase transmitter release at the frog neuromuscular junction.
Ca(2+)-dependent and -independent components of transmitter release at the frog neuromuscular junction.
Tetrahydroaminoacridine and physostigmine have opposing effects on probability of transmitter release at the frog neuromuscular junction.
Phenytoin and transmitter release at the neuromuscular junction of the frog.
Lithium ions and the release of transmitter at the frog neuromuscular junction.
Non-uniform responses to Ca2+ along the frog neuromuscular junction: effects on the probability of spontaneous and evoked transmitter release.
Interacting effects of temperature and extracellular calcium on the spontaneous release of transmitter at the frog neuromuscular junction.
A dual effect of repetitive stimulation on post-tetanic potentiation of transmitter release at the frog neuromuscular junction.
The action of verapamil on the rate of spontaneous release of transmitter at the frog neuromuscular junction.
Depression by calcium of spontaneous transmitter release at the mammalian neuromuscular junction.
Binomial analysis of quantal transmitter release at glycerol treated frog neuromuscular junctions.
Strategic location of calcium channels at transmitter release sites of frog neuromuscular synapses.
Tonic release of transmitter at the neuromuscular junction of the crab.
Spontaneous and evoked transmitter releases after concanavalin A treatment are affected differently by hypertonic low calcium solutions at frog neuromuscular junction.
Presynaptic calcium and serotonin-mediated enhancement of transmitter release at crayfish neuromuscular junction.
Evoked transmitter release increased by inorganic mercury at frog neuromuscular junction.
© 1975 Nature Publishing Group © 1975 Nature Publishing Group...
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Phenobarbital increases spontaneous transmitter release at the frog neuromuscular junction.
Temperature-sensitive aspects of evoked and spontaneous transmitter release at the frog neuromuscular junction.
Clumping and oscillations in evoked transmitter release at the frog neuromuscular junction.
Transmitter release is evoked with low probability predominately by calcium flux through single channel openings at the frog neuromuscular junction.
Changes of quantal transmitter release caused by gadolinium ions at the frog neuromuscular junction.
Inhibitory effects of cholinergic agents on the release of transmitter at the frog neuromuscular junction.
The effects of tetraphenylboron on spontaneous transmitter release at the frog neuromuscular junction.
Effect of the "calcium ionophore" A-23187 on transmitter release at the frog neuromuscular junction.
The action of lindane in accelerating the spontaneous release of transmitter at the frog neuromuscular junction.
Augmentation: A process that acts to increase transmitter release at the frog neuromuscular junction.
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