Excess beta 2 microglobulin promoting functional peptide association with purified soluble class I MHC molecules.

Peptide selection by MHC class I molecules.

Activated human T lymphocytes express MHC class I heavy chains not associated with beta 2-microglobulin.

Localization of the conformational alteration of MHC molecules induced by the association of mouse class I heavy chain with a xenogeneic beta 2-microglobulin.

A complex between the MHC class I homologue encoded by human cytomegalovirus and beta 2 microglobulin.

Dipeptides catalyze rapid peptide exchange on MHC class I molecules.

Exogenous beta 2-microglobulin is required for antigenic peptide binding to isolated class I major histocompatibility complex molecules.

Extensive peptide ligand exchange by surface class I major histocompatibility complex molecules independent of exogenous beta 2-microglobulin.

Efficient dissociation of the p88 chaperone from major histocompatibility complex class I molecules requires both beta 2-microglobulin and peptide.

Soluble class I histocompatibility antigens (s-HLA) and beta 2-microglobulin at delivery.

Lacking prognostic significance of beta 2-microglobulin, MHC class I and class II antigen expression in breast carcinomas.

Reassociation with beta 2-microglobulin is necessary for Db class I major histocompatibility complex binding of an exogenous influenza peptide.

Allele-dependent association of chicken MHC class I molecules with the invariant chain.

The role of beta-2 microglobulin in temperature-sensitive and interferon-gamma-induced exocytosis of HLA class I molecules.

Present Yourself! By MHC Class I and MHC Class II Molecules.

Peptide-binding motifs of two common equine class I MHC molecules in Thoroughbred horses.

Emerging principles for the recognition of peptide antigens by MHC class I molecules.

Peptide-independent stabilization of MHC class I molecules breaches cellular quality control.

Segregation of MHC class II molecules from MHC class I molecules in the Golgi complex for transport to lysosomal compartments.

Induction of tolerance against the arthritogenic antigen with type-II collagen peptide-linked soluble MHC class II molecules.

Assembly of MHC class I molecules analyzed in vitro.

Empty MHC class I molecules come out in the cold.

Definition of Proteasomal Peptide Splicing Rules for High-Efficiency Spliced Peptide Presentation by MHC Class I Molecules.

Automated benchmarking of peptide-MHC class I binding predictions.

Specialized functions of MHC class I molecules. I. An N-formyl peptide receptor is required for construction of the class I antigen Mta.

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Peptide selection by MHC class I molecules.

Activated human T lymphocytes express MHC class I heavy chains not associated with beta 2-microglobulin.

Localization of the conformational alteration of MHC molecules induced by the association of mouse class I heavy chain with a xenogeneic beta 2-microglobulin.

A complex between the MHC class I homologue encoded by human cytomegalovirus and beta 2 microglobulin.

Dipeptides catalyze rapid peptide exchange on MHC class I molecules.

Exogenous beta 2-microglobulin is required for antigenic peptide binding to isolated class I major histocompatibility complex molecules.

Extensive peptide ligand exchange by surface class I major histocompatibility complex molecules independent of exogenous beta 2-microglobulin.

Efficient dissociation of the p88 chaperone from major histocompatibility complex class I molecules requires both beta 2-microglobulin and peptide.

Soluble class I histocompatibility antigens (s-HLA) and beta 2-microglobulin at delivery.

Lacking prognostic significance of beta 2-microglobulin, MHC class I and class II antigen expression in breast carcinomas.

Reassociation with beta 2-microglobulin is necessary for Db class I major histocompatibility complex binding of an exogenous influenza peptide.

Allele-dependent association of chicken MHC class I molecules with the invariant chain.

The role of beta-2 microglobulin in temperature-sensitive and interferon-gamma-induced exocytosis of HLA class I molecules.

Present Yourself! By MHC Class I and MHC Class II Molecules.

Peptide-binding motifs of two common equine class I MHC molecules in Thoroughbred horses.

Emerging principles for the recognition of peptide antigens by MHC class I molecules.

Peptide-independent stabilization of MHC class I molecules breaches cellular quality control.

Segregation of MHC class II molecules from MHC class I molecules in the Golgi complex for transport to lysosomal compartments.

Induction of tolerance against the arthritogenic antigen with type-II collagen peptide-linked soluble MHC class II molecules.

Assembly of MHC class I molecules analyzed in vitro.

Empty MHC class I molecules come out in the cold.

Definition of Proteasomal Peptide Splicing Rules for High-Efficiency Spliced Peptide Presentation by MHC Class I Molecules.

Automated benchmarking of peptide-MHC class I binding predictions.

Specialized functions of MHC class I molecules. I. An N-formyl peptide receptor is required for construction of the class I antigen Mta.

Excess beta 2 microglobulin promoting functional peptide association with purified soluble class I MHC molecules.

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