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ing nine patients (four females, five males; mean age 35, range 26-44) had an identifiable cause for this finding; no patient was taking drugs other than neuroleptics. Furthermore, careful neurological examination with emphasis on cerebellar function revealed abnormalities in only three patients, two with mild dysdiadokokinesis of the non-dominant hand, and one with a slight positional tremor of both hands. Although the frequency* of gross cerebellar atrophy in other non-alcoholic populations is uncertain, a recent retrospective study of 7500 c.T. scans done at a large university hospital suggests that it is small. In their study, Allen et al. found only forty-six patients (06c,) with this finding; of these, only three were not suspected on clinical grounds of having cerebellar degeneration. There is a highly significant difference in the frequency of cerebellar atrophy between the group reported by Allan et al. and our chronic schizophrenic population (p
ing nine patients (four females, five males; mean age 35, range 26-44) had an identifiable cause for this finding; no patient was taking drugs other than neuroleptics. Furthermore, careful neurological examination with emphasis on cerebellar function revealed abnormalities in only three patients, two with mild dysdiadokokinesis of the non-dominant hand, and one with a slight positional tremor of both hands. Although the frequency* of gross cerebellar atrophy in other non-alcoholic populations is uncertain, a recent retrospective study of 7500 c.T. scans done at a large university hospital suggests that it is small. In their study, Allen et al. found only forty-six patients (06c,) with this finding; of these, only three were not suspected on clinical grounds of having cerebellar degeneration. There is a highly significant difference in the frequency of cerebellar atrophy between the group reported by Allan et al. and our chronic schizophrenic population (p
