in failure to promptly institute bed rest therapy, to delay the use of oxy¬ gen, and to remove the patient to a

lower altitude, all of which have been clearly demonstrated to be of value in the treatment of HAPE. Herbert N. Hultgren, MD Chairman, Medical Committee American

Alpine Club

Palo Alto, Calif 1.

Hultgren H, Lopez C, Lundberg E, et al: Physiologic

studies of pulmonary edema at high altitude. Circulation

29:393-408, 1964.

2. Marticorena E, Hultgren H: Treatment of high altitude pulmonary edema by bed rest alone, abstracted. Circulation 34(suppl 3):163-164, 1966. 3. Hultgren H, Billisoly J, Fails H, et al: Plasma volume changes during acute exposure to high altitude, abstracted. Clin Res 21:224, 1973.

Shower Hazard? To the Editor.\p=m-\Anew product called

"Shower Massage" is now on the market and receiving considerable adver-

tising promotion. Having purchased a unit, I found it to be quite delightful

and effective. I also found, however, that when the unit is turned to the massage setting, it does indeed massage\p=m-\especially the carotid sinus. A very high\x=req-\ intensity, pulsating water stream is delivered. In my own case, this stream was capable of considerably slowing my sinus mechanism. This could be reproduced at will. I would be concerned about the potential hazard of this device, especially in patients with sensitive carotid sinus mechanisms or cardiac arrhythmias. Syncope in a shower might prove disastrous. In older patients, massage of a partially occluded carotid artery might well result in a cerebrovascular accident. I suggest that the cardiac effect of the water massage could be easily studied by electrocardiographic monitoring of volunteer subjects in the shower. If it is indeed demonstrated that significant changes in cardiac rhythm occur, then a suitable warning should accompany the sale of the product. In any event, I would certainly ad¬ vise elderly patients against direct¬ ing the massage stream to the neck area. Sidney Richman, MD

Bloomfield, Conn

Softened Water To the Editor.\p=m-\Dr Shaper (230:130, 1974) has succinctly summarized the series of statistical studies relating

soft water and fatal heart attacks and strokes. Some of his conclusions may be erroneous in that no

naturally

distinction is made between naturally soft water and water softened by a home appliance. Naturally soft water is most often relatively acidic and has a low dissolved-solids content.1 The studies summarized by Shaper generally show that such water supplied to a community may render it susceptible to higher incidence of fatal heart attacks and strokes. Softened water, however, is more like the naturally hard water from which it is made, generally low in acidity, with relatively large quantities of dissolved solids.1 Softened water differs from the original supply in its substitution of sodium ions for virtually all calcium, magnesium, and other heavy metals. The average adult derives in excess of 90% of total daily intake of both sodium and calcium from food2 and the remainder from water. About 60% of magnesium comes from food. It is logical to conclude that the hardness of drinking water should not affect susceptibility to fatal heart attacks and that the real culprit is one of the trace constituents of water that may be associated with hardness only in a

secondary way.3 The epidemiologic studies summa¬ rized by Shaper have consistently compared naturally soft water with naturally hard water. There is no evi¬

dence that softened water can be im¬ plicated. Indeed, a study in 1971 com¬ paring naturally hard water with softened water demonstrated the re¬ verse relationship, namely, that per¬ sons using softened water suffered fatal heart attacks at a lesser rate.4 Some of the workers in the field be¬ lieve that the higher acidity of natu¬ rally soft water, compared with that of naturally hard water, leaches cad¬ mium from galvanized water pip¬ ing. M Cadmium in very small quan¬ tities has been shown to cause high blood pressure and cardiovascular dis¬ ease in test animals.5 If so, water softened in the home is not likely to be related to fatal heart attacks any more than the original hard water supply from which it is generated. A recent study (Med World News, October 1974, p 45) appears to lend further support to such a conclusion. The death rates of matched groups in Kansas City, Mo, and Kansas City, Kan, were compared. The two com¬ munities use the same water source, the Missouri River, but the Missouri city softens its water municipally. The rate of deaths from heart disease

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is 50% higher in the Kansas city, which serves its residents the harder water. The cadmium content of the Kansas community water supplied at the tap is three times that of the Mis¬ souri city and the blood of the Kansas group contained 13 times as much cadmium. The Kansas test group had significantly higher average blood pressure as well. (Municipal and do¬ mestic softening processes are known to remove cadmium and other toxic heavy metal ions that may be present in the water supply.) Two recent communications serve to further point out the confusion that exists among the many epidemiologic studies that have been re¬ ported. Three matched communities in California (Reseda, Burbank, and Downey)6 with widely varying hard¬ ness of the water supply were com¬ pared. The authors conclude that no consistent association exists between deaths from heart disease and the de¬ gree of water hardness. A similar comparison of groups in Melbourne and Brisbane, Australia,7 also yielded no consistent association. The existing data hardly warrant the strong conclusions of Dr Shaper and certainly do not implicate soft¬ ened water. However, should the con¬ sumer prefer, a hard-water line can be provided to a kitchen or bathroom tap for drinking purposes without the need to forego the benefits of soft¬ ened water. Bert Keilin, PhD, Executive Director Pacific Water Conditioning Association Irvine, Calif 1. Schroeder HA, Kraemer LA: Cardiovascular mortality, municipal water, and corrosion. Arch Environ Health 28:303-311, 1974.

2. Mickelsen 0: The nutritional and health implications of water. Proc Int Water Quality Symposium 5:35\x=req-\ 37, 1970. 3. Anderson T: Can water cause heart disease? Proc

Int Water

Quality Symposium 6:28-32,

1972.

4. Comstock GW: Fatal arteriosclerotic heart

disease, home, and socioeconomic characteristics. Am J Epidemiol 94:1-10, 1971. 5. Schroeder HA: Cadmium hypertension in rats. J Chronic Dis 18:647-656, 1965. 6. Allwright SA, Coulson A, Detels R, et al: Mortality and water hardness. Lancet 2:860-864, 1974. water hardness at

7.

Myers

D:

1:398-399, 1975.

Mortality

and water hardness. Lancet

Reply.\p=m-\Thepurpose of the short review was to indicate that we now have reasonable evidence that there is an important association between the softness and hardness of drinking water and death rates from cardiovascular disease. It is not unexpected that the manufacturers of water-softening equipment should be concerned about this association. I emphasized that we "are, however, very far from knowing what specific factors are involved or how they work." Whether In

Letter: Softened water.

in failure to promptly institute bed rest therapy, to delay the use of oxy¬ gen, and to remove the patient to a lower altitude, all of which have bee...
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