Selective cytotoxicity for transformed 3T3 cells.

Potassium transport in normal and transformed mouse 3T3 cells.

Control of growth of benzo(a)pyrene-transformed 3T3 cells.

An altered response of virally transformed 3T3 cells to ouabain.

Differential growth factor expression in transformed mouse NIH-3T3 cells.

Catecholamine hormone receptor differences identified on 3T3 and simian virus-transformed 3T3 cells.

3T3 and virus-transformed 3T3B-SV40 cells.

Estrogen enhances cytotoxicity of hyperthermia on cultured transformed cells.

c 3T3 cells.

Epidermal growth factor and the control of proliferation of Balb 3T3 and benzo[a]pyrene-transformed Balb 3T3 cells.

Resistance of in vivo-selected spontaneously transformed cells and Rous sarcoma virus-transformed cells to macrophage-mediated cytotoxicity.

Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells.

Selective killing of transformed baby hamster kidney (BHK) cells.

Spleen-cell cytotoxicity for cytomegalovirus-transformed cells. II. Inhibition by cytomegalovirus antiserum.

Exogenous glycosphingolipids suppress growth rate of transformed and untransformed 3T3 mouse cells.

Differences in intracellular distribution of plasminogen activator in growing, confluent, and transformed 3T3 cells.

Cyclic AMP-dependent protein kinases from normal and SV40-transformed 3T3 cells.

Experimental metastasis of oncogene-transformed NIH 3T3 cells in chick embryo.

Clonal dominance of select subsets of viral Kirsten ras(+)-transformed 3T3 cells during tumor progression.

Conditioned medium from ras oncogene-transformed NIH 3T3 cells induces bone resorption in vitro.

Phosphorylation but not transport of sugars is enhanced in virus-transformed mouse 3T3 cells.

The high rate of aerobic glycolysis of transformed 3T3 cells persists after cell homogenization.

pp39mos is associated with p34cdc2 kinase in c-mosxe-transformed NIH 3T3 cells.

Toxicity of metronidazole and sodium ascorbate to hypoxic SV40-transformed 3T3 cells [proceedings].

Quantitative biochemical analysis of microtubule content in normal and transformed 3T3 cells.

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Potassium transport in normal and transformed mouse 3T3 cells.

Control of growth of benzo(a)pyrene-transformed 3T3 cells.

An altered response of virally transformed 3T3 cells to ouabain.

Differential growth factor expression in transformed mouse NIH-3T3 cells.

Catecholamine hormone receptor differences identified on 3T3 and simian virus-transformed 3T3 cells.

3T3 and virus-transformed 3T3B-SV40 cells.

Estrogen enhances cytotoxicity of hyperthermia on cultured transformed cells.

c 3T3 cells.

Epidermal growth factor and the control of proliferation of Balb 3T3 and benzo[a]pyrene-transformed Balb 3T3 cells.

Resistance of in vivo-selected spontaneously transformed cells and Rous sarcoma virus-transformed cells to macrophage-mediated cytotoxicity.

Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells.

Selective killing of transformed baby hamster kidney (BHK) cells.

Spleen-cell cytotoxicity for cytomegalovirus-transformed cells. II. Inhibition by cytomegalovirus antiserum.

Exogenous glycosphingolipids suppress growth rate of transformed and untransformed 3T3 mouse cells.

Differences in intracellular distribution of plasminogen activator in growing, confluent, and transformed 3T3 cells.

Cyclic AMP-dependent protein kinases from normal and SV40-transformed 3T3 cells.

Experimental metastasis of oncogene-transformed NIH 3T3 cells in chick embryo.

Clonal dominance of select subsets of viral Kirsten ras(+)-transformed 3T3 cells during tumor progression.

Conditioned medium from ras oncogene-transformed NIH 3T3 cells induces bone resorption in vitro.

Phosphorylation but not transport of sugars is enhanced in virus-transformed mouse 3T3 cells.

The high rate of aerobic glycolysis of transformed 3T3 cells persists after cell homogenization.

pp39mos is associated with p34cdc2 kinase in c-mosxe-transformed NIH 3T3 cells.

Toxicity of metronidazole and sodium ascorbate to hypoxic SV40-transformed 3T3 cells [proceedings].

Quantitative biochemical analysis of microtubule content in normal and transformed 3T3 cells.

Selective cytotoxicity for transformed 3T3 cells.

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