Treatment with excitatory amino acids or high K+ and NMDA receptors in cerebellar granule cells.

NMDA treatment and K(+)-induced depolarization selectively promote the expression of an NMDA-preferring class of the ionotropic glutamate receptors in cerebellar granule neurones.

Nitric oxide does not mediate the neurotrophic effects of excitatory amino acids in cultured cerebellar granule neurons.

Pharmacological properties and H+ sensitivity of excitatory amino acid receptor channels in rat cerebellar granule neurones.

Pretreatment with NMDA antagonists limits release of excitatory amino acids following traumatic brain injury.

Excitatory amino acids and schizophrenia.

Carbon recycling goes full circle: fatty acids to excitatory amino acids and now excitatory amino acids to fatty acids.

Carbamazepine-induced neurotoxicity and its prevention by NMDA in cultured cerebellar granule cells.

Responses to excitatory amino acids of Purkinje cells' and neurones of the deep nuclei in cerebellar slice cultures.

Excitatory amino acids and Parkinson's disease.

Calcium channel blockers and excitatory amino acids.

Excitatory amino acids in amyotrophic lateral sclerosis.

Nonketotic hyperglycinemia: pathophysiological role of NMDA-type excitatory amino acid receptors.

Excitatory amino acid receptors and synaptic plasticity.

Excitatory amino acid receptors in epilepsy.

Distinct muscarinic receptors inhibit release of gamma-aminobutyric acid and excitatory amino acids in mammalian brain.

Excitatory amino acid-induced alterations of cytoplasmic free Ca2+ in individual cerebellar granule neurons: role in neurotoxicity.

Gliotoxic actions of excitatory amino acids.

Excitatory amino acids acting on metabotropic glutamate receptors broaden the action potential in hippocampal neurons.

Molecular characteristics of excitatory amino acid receptors.

Selective potentiation of NMDA-induced activity and release of excitatory amino acids by dynorphin: possible roles in paralysis and neurotoxicity.

Properties of non-NMDA excitatory amino acid-activated channels in isolated retinal horizontal cells.

Excitatory amino acids in epilepsy and potential novel therapies.

Quinolinate differentiates between forebrain and cerebellar NMDA receptors.

Excitatory amino acid receptor-channels in Purkinje cells in thin cerebellar slices.

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NMDA treatment and K(+)-induced depolarization selectively promote the expression of an NMDA-preferring class of the ionotropic glutamate receptors in cerebellar granule neurones.

Nitric oxide does not mediate the neurotrophic effects of excitatory amino acids in cultured cerebellar granule neurons.

Pharmacological properties and H+ sensitivity of excitatory amino acid receptor channels in rat cerebellar granule neurones.

Pretreatment with NMDA antagonists limits release of excitatory amino acids following traumatic brain injury.

Excitatory amino acids and schizophrenia.

Carbon recycling goes full circle: fatty acids to excitatory amino acids and now excitatory amino acids to fatty acids.

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Responses to excitatory amino acids of Purkinje cells' and neurones of the deep nuclei in cerebellar slice cultures.

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Excitatory amino acid receptors in epilepsy.

Distinct muscarinic receptors inhibit release of gamma-aminobutyric acid and excitatory amino acids in mammalian brain.

Excitatory amino acid-induced alterations of cytoplasmic free Ca2+ in individual cerebellar granule neurons: role in neurotoxicity.

Gliotoxic actions of excitatory amino acids.

Excitatory amino acids acting on metabotropic glutamate receptors broaden the action potential in hippocampal neurons.

Molecular characteristics of excitatory amino acid receptors.

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Treatment with excitatory amino acids or high K+ and NMDA receptors in cerebellar granule cells.

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